I've always loved this quote from comedian Groucho Marx. I'm one of those people who if given the choice would opt to live as long a life as possible. But not simply just measure of years, but the highest quality of health in those years. A want of mine is to be one of those centenarians that Dan Buettner talks about in his studies about global Blue Zones. To be 100 and leading an active, vibrant life full of purpose, passion and zip, that sounds pretty awesome to me. Fortunately for me, emerging mitochondrial science is on my side and helping us better understand the relationship between mitochondria, aging and disease.
In July I was approached by MitoQ to personally trial and review their award-winning Serum and Antioxidants. The results I felt were notable. Not only did I feel a boost in energy, I also found that over the 4-month period I saw a noticeable improvement in stress levels and workout recovery times – both decreased. Was it a placebo effect? I wondered that myself but I'm not willing to stop taking the product to find out.
Can a supplement help me live to be 100?
This follow-up post is to give more info on MitoQ, mitochondria and more info about the emerging scientific studies and theories, which are grabbing not only the attention of consumers like you and me, but governing bodies too. Most notably, MitoQ has been chosen by the US government’s National Institute of Aging as part of a major research project on anti-aging.
“The National Institute of Aging is part of the US National Institute of Health, an organization that invests over $30 billion per annum into health research. MitoQ was chosen for the testing program because MitoQ shows significant potential to delay or decelerate the aging process and improve general health,” said Greg Macpherson, CEO of MitoQ. NIA's Interventions Testing Program (ITP) is an internationally renowned anti-aging testing program funded by the US Government. The results will be revealed in 2020.
So, still not sure what I'm talking about?
No worries. Honestly before I started doing some research on mitochondria, mitochondrial disease and such, I felt like I was back in high school biology, trying to cram for a mid-term. Luckily I have the internet today instead of the library and microfiche I relied on back in high school.
Today there are organizations like the United Mitochondrial Disease Foundation and Mitochondria Research and Medicine Society, which are dedicated to mitochondrial research.
Part 1: What is Mitochondria?
The best high-school, easy to understand definition of mitochondria I found was care of Wikipedia.org:
“Mitochondria are organelles, or parts of a cell. They make most of the cell's supply of adenosine triphosphate (ATP), a molecule that cells use as a source of energy. Their main job is this energy conversion. They oxidize glucose to give energy for the cell. The process makes ATP, and is called cellular respiration. In addition to supplying cellular energy, mitochondria are involved in a range of other processes, such as signaling, cellular differentiation, cell death, as well as the control of the cell division cycle and cell growth.”
The takeaway: Mitochondria are responsible for creating more than 90% of the energy needed by the body to sustain life and support growth.
Part 2: The Role of Mitochondria in Aging and Disease
There are two main theories about aging in humans:
- That we are genetically programmed to live a certain amount of time. And,
- That over time our body and (mitochondrial) DNA is damaged until we can no longer function properly (see telomere shortening and oxidative stress).
Whether we wear out over time or are programmed to only live a certain amount of time, science would keep up that IF we can change our genes responsible for aging and slow or delay the damage of our DNA, we could live longer. And there's a couple studies that support these anti-aging theories.
In a recent issue of Nature, Swedish scientists observed that tiny changes in cellular mitochondrial DNA is responsible for aging (well at least in mice). They found that mice that had been bred to have mutations (i.e. dysfunctional) to their mitochondrial DNA only lived on average 48 weeks, whereby normal lab mice tend to live 2 years. That's significant!
Furthermore, a study involving worms found that through gene manipulation they were able to increase their lifespan 5-fold. That would be 400 to 500 in human years! WHOA!
And how does mitochondrial disease affect us:
In a person with mitochondrial disease, the mitochondria are failing and cannot convert food and oxygen into life-sustaining energy. For many, mitochondrial disease is an inherited genetic condition, while for others the body's mitochondria can be affected by other environmental factors.
Diseases of the mitochondria seem to cause the most damage to cells of the brain, heart, liver, skeletal muscles, kidney and the endocrine and respiratory systems.
Depending on which cells are affected, symptoms may include loss of motor control, muscle weakness and pain, gastrointestinal disorders and swallowing difficulties, poor growth, cardiac disease, liver disease, diabetes, respiratory complications, seizures, visual/hearing problems, lactic acidosis, developmental delays and susceptibility to infection. – UMDF
But, until science is able to definitively tell us how to offset again, we're all faced with mitochondrial disease to some level.
A great discussion from Dr. David Sinclair on the Role of Mitochondria in Aging and Disease:
Part 3: What can we do to support healthy mitochondria and delay oxidative stress?
It's actually quite simple to improve our cellular health, many of which are smart lifestyle decisions made daily: exercise, eat smart, don't smoke, get adequate sleep, keep well hydrated and supplement with mitochondrial-targeted anti-oxidants.
These are a brand new type of antioxidant, able to deliver a payload of free radical defense directly into the mitochondria. Discovered at Otago University (Dunedin, New Zealand), they are considered to be a significant advance in antioxidant technology.
Until this discovery, no-one had been able to restore the mitochondrial antioxidant defense system in a meaningful way. This is because the mitochondrial membrane is particularly impermeable to substances. Many supplements are simply not able to get to where they need to be—inside the mitochondria in your cells.
Seems simple to me… but don't confuse simple with easy. If it was easy we'd all be doing it, right?
Part 4: Why MitoQ is a good bet as an investment in a healthier, longer life
Developed at Otago University, MitoQ delivers antioxidants into the mitochondria, right where they are needed.
By restoring the mitochondrial free radical defense system, we achieve the double benefit of both restoring mitochondrial function and reducing free radical leakage into the cell. This supports optimal mitochondrial function and results in improved energy production for the cell. Our cells and organs get an energy boost, and that helps them run like they did when they were younger.
But instead of reading all the supportive studies, check out this short video about MitoQ and how it plays in restoring mitochondrial function and cellular health:
For more reading and research check out the free e-book care of MitoQ. Download your copy here.
As a special offer for DaiManuel.com readers and tribe members, MitoQ has offered up a 20% discount on your first order.
I don't know about you, but I'm going to do my best to live to a 100 years old. Daily exercise, eat less than I need, sleep well and stay well hydrated (and, of course, take my MitoQ).
Seems EASY to me, how about you?
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This is a sponsored conversation written by me on behalf of MitoQ. The opinions and text are all mine.
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